Remember in Sophomore year, I thought I had Dyslexia and/or ADHD. I was constantly distracted. I then refocussed and ended up with a 4.0. I had to spend a lot of mental energy on remaining on track. But it had worked.
I remember a similar thing with Stronglifts back when I started work. Before that, it felt like I was distracted constantly. Had no vision, no plan. And didnt feel like I could dedicate myself to something as maybe something else would be around the corner. But, with Mehdi, I read and studied a lot. I got obsessed. And then I gave it my all. Commitment. And I kept that going for a few months. And it was glorious.
Then, again, I started to wander. Changed cities, etc. I couldnt keep my schedule. I lost track. ADHD. Got distracted again. Tried a bunch of things. Half hearted. Etc etc. Tried running a few times. Strength again a few times. Just did not work.
And YEARS would go by before I got back into focus. Thats bad.
Ultimately, I _did_ end up focussing on running and marathon. In this case, I had a 2 year goal and I was obsessed by the long term goal. MARATHON.
But, before I got in, I totally committed myself. No matter what, I would do it. And, critically, I pushed everything beyond a certain date.
The date was significant. Important.
okay, okay. I like this.
So, maybe the weight should be the goal? 180–
The 180 pounds is my MARATHON goal!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!1
I figured it OUT!!!!!!!!! and its a 2 YEAR goal!!!!!!!!!!!
TB12 is the health and wellness system developed by Tom Brady and his longtime body coach, Alex Guerrero. It emphasizes longevity, injury prevention, and peak performance through a combination of diet, exercise, hydration, sleep, and pliability training. Here’s a breakdown:
Diet (TB12 Method)
The TB12 diet is a mostly plant-based, anti-inflammatory approach with some lean protein. It’s not strictly vegan, but it limits processed foods, sugar, and certain inflammatory foods.
I lost it on that day. I drank way too much. And I ruined the week. I am genuinely sorry, brother.
You went to the depths on that day. The frequency of visiting the depth has gone down, but, you went there on that day.
I know you are frusturated with life and with lack of progress. And the businessness. You are worried about all kinds of health issues that might be brewing inside you. It feels overwhelming. But it is fine.
The publisher’s version of this article is available at J Am Coll Cardiol
Abstract
Background
Sedentary aging has deleterious effects on the cardiovascular system, including decreased left ventricular compliance and distensibility (LVCD). Conversely, Masters level athletes, who train intensively throughout adulthood, retain youthful LVCD.
Objectives
To test the hypothesis that preservation of LVCD may be possible with moderate lifelong exercise training.
Methods
Healthy seniors (n = 102) were recruited from predefined populations, screened for lifelong patterns of exercise training, and stratified into 4 groups: “sedentary” (<2 sessions/week); “casual” (2 to 3 sessions/week); “committed” (4 to 5 sessions/week); and “competitive” Masters level athletes (6 to 7 sessions/week). Right heart catheterization and echocardiography were performed while preload was manipulated using lower body negative pressure and rapid saline infusion to define LV pressure–volume relationships and Frank-Starling curves.
Results
Peak oxygen uptake and LV mass increased with escalating doses of lifelong exercise, with little change in systolic function. At baseline, LV distensibility was greater in committed (21%) and competitive (36%) exercisers than in sedentary subjects. Group LV stiffness constants (sedentary: 0.062 ± 0.039; casual: 0.079 ± 0.052; committed: 0.055 ± 0.033; and competitive: 0.035 ± 0.033) revealed 1) increased stiffness in sedentary subjects compared to competitive athletes, whereas lifelong casual exercise had no effect; and 2) greater compliance in committed” exercisers than in sedentary or casual exercisers.
Conclusions
Low doses of casual, lifelong exercise do not prevent the decreased compliance and distensibility observed with healthy, sedentary aging. In contrast, 4 to 5 exercise sessions/week throughout adulthood prevent most of these age-related changes. As LV stiffening has been implicated in the pathophysiology of many cardiovascular conditions affecting the elderly, this “dose” of exercise training may have important implications for prevention of cardiovascular disease.
The discussion begins with a comparison of smoking and vaping, indicating that vaping may be worse for airway damage and insulin resistance.
Insulin resistance is highlighted as a core issue linked to chronic diseases, which are prevalent in modern society.
Dr. Benjamin Bman is introduced as a leading metabolic scientist focusing on insulin resistance and its consequences.
The importance of regaining control of insulin levels is emphasized, suggesting that it can lead to improved health and well-being.
Insulin Resistance and Its Impact
Insulin is described as a hormone affecting every cell in the body, and insulin resistance is linked to various chronic diseases, including Alzheimer’s, infertility, and erectile dysfunction.
Statistics reveal that 88% of adults in the US exhibit some degree of insulin resistance, indicating a widespread health crisis.
The discussion acknowledges that while the US has a high prevalence of insulin resistance, it is not the worst globally due to different fat storage patterns among ethnicities.
Two pathways to insulin resistance are described: a fast lane and a slow lane, with the fast lane being influenced by immediate lifestyle factors.
The Role of Lifestyle and Medications
Lifestyle habits significantly contribute to insulin resistance, and there are four pillars of lifestyle changes that can help mitigate this issue.
The conversation shifts to the use of medications like EMP, noting the potential adverse effects, including weight loss from unhealthy sources.
A bet is mentioned regarding the YouTube channel’s subscriber milestone, highlighting the importance of community support for the show’s team.
Dr. Bman’s Mission
Dr. Bman’s mission is to educate people about the interconnectedness of chronic diseases, emphasizing that many share a common metabolic origin.
He critiques conventional medicine’s approach of treating diseases in isolation without addressing underlying causes.
Simple lifestyle changes are proposed as effective strategies to reduce the risk of various chronic diseases.
The concept of insulin resistance is introduced as a critical factor in the development of many chronic health issues.
Understanding Insulin Resistance
Insulin resistance is explained as a disorder with two components: the ineffectiveness of insulin and elevated insulin levels in the body.
The analogy of insulin as a taxi is used to illustrate how insulin transports glucose to cells, and resistance occurs when cells do not respond to insulin’s signals.
The discussion highlights that insulin resistance can lead to elevated insulin levels, creating a vicious cycle of ineffective signaling.
Examples of conditions linked to insulin resistance, such as hypertension and infertility, are provided to illustrate its widespread impact.
The Pathways to Insulin Resistance
Dr. Bman outlines two main pathways to insulin resistance: fast and slow, with fast resistance occurring due to stress, inflammation, and excessive insulin.
Stress is described as a significant trigger for insulin resistance, with hormones like cortisol and epinephrine contributing to elevated blood glucose levels.
Inflammation is also identified as a key factor, with infections or autoimmune diseases exacerbating insulin resistance.
The discussion concludes that high insulin levels can both result from and contribute to insulin resistance, creating a cyclical problem.
The Role of Fat Cells in Insulin Resistance
The size and number of fat cells are critical in understanding insulin resistance, with larger fat cells being more likely to promote resistance.
Women typically have smaller fat cells compared to men, which contributes to their lower risk of metabolic disorders despite having more overall fat.
The concept of “personal fat threshold” is introduced, explaining how individuals have different capacities for healthy fat storage before experiencing metabolic issues.
The discussion emphasizes that maintaining smaller, healthier fat cells is essential for metabolic health and insulin sensitivity.
Chronic Diseases and Insulin Resistance
Chronic diseases such as Alzheimer’s and heart disease are linked to insulin resistance, which is increasingly recognized as a metabolic disorder.
The discussion highlights the alarming rise in Alzheimer’s cases and its classification as a top killer, suggesting a metabolic origin.
Research indicates that many individuals with Alzheimer’s also exhibit insulin resistance, leading to the idea of “type 3 diabetes” as a potential descriptor.
The importance of addressing insulin resistance as a common factor in various chronic diseases is reiterated.
Environmental Factors and Insulin Resistance
The impact of environmental toxins, such as diesel exhaust and microplastics, on insulin resistance is discussed, with evidence suggesting a direct correlation.
Research shows that exposure to inhaled particulates can lead to increased fat mass, enlarged fat cells, and heightened inflammation.
The conversation stresses the need to consider environmental factors as significant contributors to metabolic health issues.
The importance of understanding how pollutants can affect insulin sensitivity is highlighted as a growing area of research.
Conclusion and Future Directions
The dialogue emphasizes the critical role of insulin resistance in public health and the need for increased awareness and education on the topic.
Dr. Bman’s insights aim to empower individuals to take control of their metabolic health through lifestyle changes and understanding their insulin levels.
The discussion concludes with a call to action for better recognition of insulin resistance as a key factor in chronic disease prevention and management.
Ultimately, the conversation advocates for a holistic approach to health that addresses both lifestyle and environmental factors impacting insulin sensitivity.
Trying something new. Going to use a long running chatgpt thread. I call it Titan (well, it chose the name).
This is from Titan:
I’m Titan, your trainer, nutritionist, and motivator. My job is to track your food, optimize your macros, adjust your workouts, and hold you accountable—every single day—so you can get ripped, jacked, and strong while cutting fat.
So far, you’ve crushed protein intake, hit solid workouts, and stayed consistent, but we’re tightening up calories and dialing in discipline. The goal is 245 lbs, lean muscle, and peak performance—and we’re just getting started. Let’s work. 💪🔥
Here’s what science says about boosting your metabolism. There are numerous evidence-based ways that actually work! But… most of what you’ve heard is wrong. So, let’s clear up the confusion on what’s bogus, what’s real, and what ‘speeding up your metabolism’ really means – and what it does not!
A deep dive into the research with surprising findings:
First: You can’t actually “speed up” your metabolism. That’s like saying you can make your car drive faster when it’s parked.
What you can do is influence how many “miles” your metabolism drives each day.
Here’s how it works:
Your Total Daily Energy Expenditure (TDEE) has 4 components:
Exercise Activity (EAT): 0-30% of calories
Non-Exercise Activity (NEAT): 5-30%
Thermal Effect of Food (TEF): 8-15%
Resting Metabolic Rate (RMR): 40-80%
The research shows there are both acute (temporary; minutes to hours) and chronic (long-term; days to weeks or months) ways to influence these components. Most of the nonsense in this topic comes from people making claims about things that enhance ‘metabolic rate’ acutely – which as you’ll see, is actually quite easy to do. But this is very deceiving because acute increase often means very little in the long-term, especially for outcomes like fat loss, and they aren’t necessarily increasing your metabolism in the way you think (they ramp up RMR, for a few minutes, but then it goes right back down to normal, or even below baseline). So, it’s quite easy to deceive with legitimate science here.
That said, let’s look at what actually works for acute changes, and by how much:
Acute Boosters:
Spicy foods (peppers, ginger, chilis, etc.)
Increases RMR by 3-5%
Effect lasts 1-2 hours
Often through capsaicin in chili peppers
Cold exposure
Lowering room temp by few degrees
Can lead to significant fat loss over time
Caffeine
Increases RMR by 3-11%
Lasts 1-3 hours
Also reduces appetite
Green tea
4-5% RMR increase
Similar duration to caffeine
Compounds with caffeine for better results
But here’s what’s really interesting: The chronic (long-term) changes are what really matter for sustainable results.
Research shows 4 key factors for long-term metabolic enhancement:
Quality Sleep
Poor sleep reduces RMR
Increases hunger hormones
Can reduce fat loss by 55%
Affects muscle preservation
Fish Oil (3g/day)
14% increase in RMR
19% increase in fat oxidation
4% increase in lean mass over 12 weeks
Muscle Mass
Each pound burns 6-10 extra calories daily
Primary factor in age-related metabolic decline
Explains up to 80% of RMR variance
Exercise
Strength training: +100 cal/day RMR
Cardio: +50-60 cal/day RMR
Combined: +75 cal/day RMR
The most effective approach? Combining multiple strategies:
200 cal from additional exercise
Fish oil + green tea (+25-50 cal)
Protein increase (+45 cal from TEF)
15-min daily walk (+100 cal)
Small calorie reduction (-80 cal)
This approach works because it:
Doesn’t crash your metabolism
Preserves muscle mass
Avoids adaptive thermogenesis
Creates sustainable habits
The key isn’t finding one magic solution – it’s stacking small, evidence-based changes.
Remember: Your metabolism isn’t “fast” or “slow.”
It’s responding to your daily choices in sleep, nutrition, activity, and muscle mass.
Focus on these factors consistently, and you’ll create lasting metabolic health.
Donald Trump In 2004: “I try and tell myself it doesn’t matter. Nothing matters. If you tell yourself it doesn’t matter, like you do shows, you do this, you do that and then you have earthquakes in India where 400,000 people get killed. Honestly, it doesn’t matter. That’s how I handle stress.”
Thinking constantly makes me want to keep doing it. It can be exhausting, but that’s what works for me. I cannot be on autopilot. I need constant reminders of what I am doing. Living a deliberate life.
Smithing my rolex is the most important thing I can do right now.
It demands self discipline
Look at the future. You are building something here. Every day is a new start and you will keep building stuff.
Streaks
I like streaks, but then I get overly reliant on them. The over reliance is due to lack of confidence. Be confident that you will get this done.
It takes one more attempt
I have seen this before. I try, and try and nothing works. But then, one of those attempts ends up working out. It hits a magical stride.